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Infrequent hypermethylation of WIF-1 promoter in BCR/ABL-negative myeloproliferative disorders.
The Tokai journal of experimental and clinical medicine

The JAK2(V617F) tyrosine kinase mutation in myeloproliferative disorders: Summary of published literature and a perspective.
Current hematologic malignancy reports

JAK2 V617F patients with essential thrombocythemia present with clinical features of polycythemia vera.
Leukemia & lymphoma

[Tyrosine kinase JAK2V617F mutation in human myeloproliferative disorders].
Zhongguo shi yan xue ye xue za zhi / Zhongguo bing li sheng li xue hui = Journal of experimental hematology / Chinese Association of Pathophysiology

Quantitation of the JAK2V617F mutation in microdissected bone marrow trephines: equal mutational load in myeloid lineages and rare involvement of lymphoid cells.
The American journal of surgical pathology

Absence of JAK2 V617F mutation in thalassemia intermedia patients.
Molecular biology reports

Activated STAT1 and STAT5 transcription factors in extramedullary hematopoietic tissue in a polycythemia vera patient carrying the JAK2 V617F mutation.
International journal of hematology

Bone marrow phospho-STAT5 expression in non-CML chronic myeloproliferative disorders correlates with JAK2 V617F mutation and provides evidence of in vivo JAK2 activation.
The American journal of surgical pathology

The myeloproliferative disorder-associated JAK2 V617F mutant escapes negative regulation by suppressor of cytokine signaling 3.
Blood

The chronic myeloproliferative disorders and mutation of JAK2: Dameshek's 54 year old speculation comes of age.
Best practice & research. Clinical haematology

[Novel method in diagnosis of chronic myeloproliferative disorders--detection of JAK2 mutation].
Orvosi hetilap

Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders.
Lancet

[Abnormal activation of tyrosine kinases and its role in the pathogenesis of hematological malignancies - review].
Zhongguo shi yan xue ye xue za zhi / Zhongguo bing li sheng li xue hui = Journal of experimental hematology / Chinese Association of Pathophysiology

Widespread occurrence of the JAK2 V617F mutation in chronic myeloproliferative disorders.
Blood

The role of JAK2 V617F mutation, spontaneous erythropoiesis and megakaryocytopoiesis, hypersensitive platelets, activated leukocytes, and endothelial cells in the etiology of thrombotic manifestations in polycythemia vera and essential thrombocythemia.
Seminars in thrombosis and hemostasis

Detection of the activating JAK2 V617F mutation in paraffin-embedded trephine bone marrow biopsies of patients with chronic myeloproliferative diseases.
The Journal of molecular diagnostics : JMD

Chronic idiopathic myelofibrosis: clinicopathologic features, pathogenesis, and prognosis.
Archives of pathology & laboratory medicine

[Diagnostic algorithm in chronic myeloproliferative diseases (CMPD)].
Medizinische Klinik (Munich, Germany : 1983)

A role for JAK2 mutations in myeloproliferative diseases.
Annual review of medicine

Advances in the therapy of chronic idiopathic myelofibrosis.
The oncologist

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